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organs  that  could  possibly  be  affected  and  there  was  a  detailed  rep ort  for
                ever y organ of ever y subject. e  procedures in e  “G” Report were  almost

                certainly  vivisections.  is  was  due  to  there  being  multiple  points  in  which

                the author referred to information about the  disease’s progression that could

                only  be  attained  by  having  dissected  the  subject  before  they  died—days  or

                weeks before the disease evolved to its  nal, let hal, stage. However, similar to

                the  “A”  Report,  this  report  lacks  any  speci c  description  of  the  vivisection
                procedures as it is more of a report of the  results. On page 109 of e Report

                of   “G,”   the   author   states,   “I   cannot   explain   ‘the   met hods   of   infection   and

                clinical  symptoms’  in  detail,  while    I  have   not  received  thes e   records.”  is

                shows  that  the  author,  though  present  at  the  vivisections  of  the  subjects  (in

                order   to   even   create   the   report),   did   not   oversee   the   entire   process   of
                infecting   and   monitoring     the   subjects.   is   explains   why   there   is   so   little

                information  on  the  details  or  circumstances  of  the  vivisections;  the  author

                was   most    likely   not   present   for   some   portion   of   the   exper iment   likely

                performed by a low level technician.



                From    detailed   notes   on   the   reported   results   of   the   disease,   the   e  ects   of

                glanders disease, include:




                      Heart:  Interstitial  edema  in  the  subendocardial  tissues  (swelling  in  the

                       uid- lled  space  of  muscular  walls  of  the  heart  which  contain  ner ves
                      and    impulse-conducting        system     that   allows   it   to   beat),   miliar y

                      glanders-knots  (scabs  and  rashes  in  the  inner  tissues  of  the  heart,  some

                      of   which   inhibit   the    ow   of   blood   in   the   heart   through   swelling),

                      degeneration  in  the  cartilage  of  the  heart  (weakening  and  deter ioration

                      of  the  heart,  reducing  its  ability  to  circulate  blood),  Rheumatoid-knots
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